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Boston Scientific Corporation Message Board

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  • juststent3195 juststent3195 Oct 26, 2006 6:59 PM Flag

    the evening world news

    Cypher work two ways: in the G1 phase, cytostatically turning off the cell as it is receiving cellular mediators to begin restonic proliferation. Turns the cell in a hibernated state. Unlike Taxol that works in the S phase of the cell cycle, terminating the cell. Cypher also works in the inflammatory response.

    The drug is completely eluded after 90 days on a Cypher stent (IFU), while 90% of Taxol on Taxus stays within the polymer indefinitely (DFU). This can be a hypothesis as to way their is an increase in non-Q wave MI in overlapping Taxus stents (DFU) and not Cypher. May also provide insite as to the potential of higher late events with Taxus. Half of Cypher's drug is gone in 8 days, yet you grouped all DES in the category of long drug release mechanisms. Drug does need to be there more than 90 days, because most of the inflammation and resulting health (restonsis) stage are complete by then.

    You did not comment on my statement about the length of time stents have been used and their durability, yet you recommend rethinking the hole procedure completely.

 
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